Anonymous
(ID: Nxonl+Y/)
9/11/2025, 4:11:25 PM
No.515510864
[Report]
>>515512112
>>515513104
>>515516344
9/11/2025, 4:11:25 PM
No.515510864
[Report]
>>515512112
>>515513104
>>515516344
Timeline of events of the killing of Charlie Kirk
He didn't even realize he was shot.
>Moment of Impact (0–1 second):
The bullet struck the left side of the neck, destroying critical vascular and neural structures.
Likely injuries: carotid artery transection, jugular vein rupture, severe disruption of vagus nerve and surrounding soft tissue.
Possible concurrent spinal cord trauma (suggested by immediate limp posture and lack of protective movement).
Immediate massive haemorrhage occurred, estimated at >1 liter within the first second.
>Immediate Consequences (within 1–3 seconds):
Cerebral perfusion collapse: With the carotid artery destroyed, blood supply to the brain fell catastrophically.
This caused instantaneous loss of consciousness, comparable to syncope but irreversible.
The limp body posture and absence of reflexive neck-grabbing indicate no cortical awareness at the moment of impact.
Some observers noted decorticate-like posturing of the right hand, which can occur in severe CNS injury.
>Short-Term Phase (3–15 seconds):
Continuous, high-pressure external bleeding ensured that blood pressure dropped to non-survivable levels.
Even with the Circle of Willis compensating, the magnitude of vascular destruction meant no effective collateral circulation could sustain consciousness.
Neurologically: brain anoxia and rapid neuronal failure began almost immediately.
>Terminal Phase (15–60 seconds):
With ongoing exsanguination, the body entered irreversible hypovolemic shock.
Loss of spinal cord integrity (if involved) would also paralyze autonomic and motor function.
Death was essentially instantaneous, though cardiac electrical activity may have continued for a minute or two without cerebral function.
>Moment of Impact (0–1 second):
The bullet struck the left side of the neck, destroying critical vascular and neural structures.
Likely injuries: carotid artery transection, jugular vein rupture, severe disruption of vagus nerve and surrounding soft tissue.
Possible concurrent spinal cord trauma (suggested by immediate limp posture and lack of protective movement).
Immediate massive haemorrhage occurred, estimated at >1 liter within the first second.
>Immediate Consequences (within 1–3 seconds):
Cerebral perfusion collapse: With the carotid artery destroyed, blood supply to the brain fell catastrophically.
This caused instantaneous loss of consciousness, comparable to syncope but irreversible.
The limp body posture and absence of reflexive neck-grabbing indicate no cortical awareness at the moment of impact.
Some observers noted decorticate-like posturing of the right hand, which can occur in severe CNS injury.
>Short-Term Phase (3–15 seconds):
Continuous, high-pressure external bleeding ensured that blood pressure dropped to non-survivable levels.
Even with the Circle of Willis compensating, the magnitude of vascular destruction meant no effective collateral circulation could sustain consciousness.
Neurologically: brain anoxia and rapid neuronal failure began almost immediately.
>Terminal Phase (15–60 seconds):
With ongoing exsanguination, the body entered irreversible hypovolemic shock.
Loss of spinal cord integrity (if involved) would also paralyze autonomic and motor function.
Death was essentially instantaneous, though cardiac electrical activity may have continued for a minute or two without cerebral function.
