What are your biggest icks?
>balding
>glasses
>mouth breathing
>bad hygiene
>cluster B mental illness
4chan Search
16 results for "c03a5efd4b1f46acbb68f06f28d82736"
Is this why so many zoomers have the broccoli cut?
Why do white guys, jeet indians, self hating east asians/southeast asians, muslims, browns, mixed race mutts and spics watch bbc porn all day? i noticed the more they admire white supremacy the more they watch bbc porn lol. i can understand blacks/nigs watching bbc porn but why dont you guys just watch porn of men in your own race fucking girls.
why do white socks look zesty
/balding/ general
/balding/ and /finasteride/ general - hair is love, hair is life!
Sticky: http://tuxbell.com/index.php/Balding
Reminder that if you can clearly see down to your scalp when your hair is wet, your hair is thinning, lots of small new hairs sprouting at the hairline or crown is a sign of hair recession.
Holy trinity:
Tabs: https://en.wikipedia.org/wiki/Finasteride
Lotion: https://en.wikipedia.org/wiki/Minoxidil
Shampoo: https://en.wikipedia.org/wiki/Ketoconazole
Finasteride/Dutasteride are 5-alpha reductase inhibitors that prevent free T from being converted to DHT, a byproduct of testosterone that serves no purpose after puberty but to choke out your hair follicles and make them thin out and die in a few short years, a condition known as androgenic alopecia.
5ar inhibitors prevent your hair from getting any worse, mild regrowth is expected, rapid onset diffuse thinners see a significant recovery on fin alone. Can be taken orally or topically.
5ARIs are not dosage dependent, taking as little as as 0.2mg daily fin can reduce serum DHT by 68%, while 5mg reduces by 72%.
0.5mg daily dut reduces serum DHT by 95%, but is considered a second line treatment, and can be more expensive and harder to get prescribed. Start with fin.
Fin has been used to treat benign prostatic hyperplasia (BPH) for 30+ years, it's very well studied and is tolerated well in almost all men, however, there's a 1-2% risk of erectile issues in controlled studies.
These can be avoided by reducing dosage, spacing out dosage, or stopping the drug. Fin takes days to fully bio-accumulate, the half life of fin is about 8 hours, adverse effects occuring within minutes of starting fin or persisting long after stopping fin are not real and have no basis in reality. Please note that dut has a much higher half-life of 5 weeks, if you're prone to fin sides, dut may cause them for far longer.
Refer to https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3481923/ before responding to PFS trolls.
(continued on next post)
Sticky: http://tuxbell.com/index.php/Balding
Reminder that if you can clearly see down to your scalp when your hair is wet, your hair is thinning, lots of small new hairs sprouting at the hairline or crown is a sign of hair recession.
Holy trinity:
Tabs: https://en.wikipedia.org/wiki/Finasteride
Lotion: https://en.wikipedia.org/wiki/Minoxidil
Shampoo: https://en.wikipedia.org/wiki/Ketoconazole
Finasteride/Dutasteride are 5-alpha reductase inhibitors that prevent free T from being converted to DHT, a byproduct of testosterone that serves no purpose after puberty but to choke out your hair follicles and make them thin out and die in a few short years, a condition known as androgenic alopecia.
5ar inhibitors prevent your hair from getting any worse, mild regrowth is expected, rapid onset diffuse thinners see a significant recovery on fin alone. Can be taken orally or topically.
5ARIs are not dosage dependent, taking as little as as 0.2mg daily fin can reduce serum DHT by 68%, while 5mg reduces by 72%.
0.5mg daily dut reduces serum DHT by 95%, but is considered a second line treatment, and can be more expensive and harder to get prescribed. Start with fin.
Fin has been used to treat benign prostatic hyperplasia (BPH) for 30+ years, it's very well studied and is tolerated well in almost all men, however, there's a 1-2% risk of erectile issues in controlled studies.
These can be avoided by reducing dosage, spacing out dosage, or stopping the drug. Fin takes days to fully bio-accumulate, the half life of fin is about 8 hours, adverse effects occuring within minutes of starting fin or persisting long after stopping fin are not real and have no basis in reality. Please note that dut has a much higher half-life of 5 weeks, if you're prone to fin sides, dut may cause them for far longer.
Refer to https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3481923/ before responding to PFS trolls.
(continued on next post)
/balding/ general
/balding/ and /finasteride/ general - hair is love, hair is life!
Sticky: http://tuxbell.com/index.php/Balding
Reminder that if you can clearly see down to your scalp when your hair is wet, your hair is thinning, lots of small new hairs sprouting at the hairline or crown is a sign of hair recession.
Holy trinity:
Tabs: https://en.wikipedia.org/wiki/Finasteride
Lotion: https://en.wikipedia.org/wiki/Minoxidil
Shampoo: https://en.wikipedia.org/wiki/Ketoconazole
Finasteride/Dutasteride are 5-alpha reductase inhibitors that prevent free T from being converted to DHT, a byproduct of testosterone that serves no purpose after puberty but to choke out your hair follicles and make them thin out and die in a few short years, a condition known as androgenic alopecia.
5ar inhibitors prevent your hair from getting any worse, mild regrowth is expected, rapid onset diffuse thinners see a significant recovery on fin alone. Can be taken orally or topically.
5ARIs are not dosage dependent, taking as little as as 0.2mg daily fin can reduce serum DHT by 68%, while 5mg reduces by 72%.
0.5mg daily dut reduces serum DHT by 95%, but is considered a second line treatment, and can be more expensive and harder to get prescribed. Start with fin.
Fin has been used to treat benign prostatic hyperplasia (BPH) for 30+ years, it's very well studied and is tolerated well in almost all men, however, there's a 1-2% risk of erectile issues in controlled studies.
These can be avoided by reducing dosage, spacing out dosage, or stopping the drug. Fin takes days to fully bio-accumulate, the half life of fin is about 8 hours, adverse effects occuring within minutes of starting fin or persisting long after stopping fin are not real and have no basis in reality. Please note that dut has a much higher half-life of 5 weeks, if you're prone to fin sides, dut may cause them for far longer.
Refer to https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3481923/ before responding to PFS trolls.
(continued on next post)
Sticky: http://tuxbell.com/index.php/Balding
Reminder that if you can clearly see down to your scalp when your hair is wet, your hair is thinning, lots of small new hairs sprouting at the hairline or crown is a sign of hair recession.
Holy trinity:
Tabs: https://en.wikipedia.org/wiki/Finasteride
Lotion: https://en.wikipedia.org/wiki/Minoxidil
Shampoo: https://en.wikipedia.org/wiki/Ketoconazole
Finasteride/Dutasteride are 5-alpha reductase inhibitors that prevent free T from being converted to DHT, a byproduct of testosterone that serves no purpose after puberty but to choke out your hair follicles and make them thin out and die in a few short years, a condition known as androgenic alopecia.
5ar inhibitors prevent your hair from getting any worse, mild regrowth is expected, rapid onset diffuse thinners see a significant recovery on fin alone. Can be taken orally or topically.
5ARIs are not dosage dependent, taking as little as as 0.2mg daily fin can reduce serum DHT by 68%, while 5mg reduces by 72%.
0.5mg daily dut reduces serum DHT by 95%, but is considered a second line treatment, and can be more expensive and harder to get prescribed. Start with fin.
Fin has been used to treat benign prostatic hyperplasia (BPH) for 30+ years, it's very well studied and is tolerated well in almost all men, however, there's a 1-2% risk of erectile issues in controlled studies.
These can be avoided by reducing dosage, spacing out dosage, or stopping the drug. Fin takes days to fully bio-accumulate, the half life of fin is about 8 hours, adverse effects occuring within minutes of starting fin or persisting long after stopping fin are not real and have no basis in reality. Please note that dut has a much higher half-life of 5 weeks, if you're prone to fin sides, dut may cause them for far longer.
Refer to https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3481923/ before responding to PFS trolls.
(continued on next post)
>ex's friend calls me multiple times a day
>despite being blocked for 3+ months after breaking up with my ex
What the fuck? When will he give up?
Fucking zoomers are so persistent and weird. Learn to take a fucking hint, Jamie, I know you post here
>despite being blocked for 3+ months after breaking up with my ex
What the fuck? When will he give up?
Fucking zoomers are so persistent and weird. Learn to take a fucking hint, Jamie, I know you post here
What causes baldness really? Creatine? Roids? Stress?
the fuck are these zoomies eating
the fuck are these zoomies eating
the fuck are these zoomies eating
this type of zoomer balding seems really unnatural
usually we see slow recession of the hair line in an M shape but the one's in this webm bald like africans. it's like a rounded shape so you have to wonder if it's even testostorone or DHT related. they remind me of the balding marvel basedcuck memes so it has to be something related to estrogen.
personally
do you feel like your gamer skills have declined with age?
/balding/ general
/balding/ and /finasteride/ general - hair is love, hair is life!
Sticky: http://tuxbell.com/index.php/Balding
Reminder that if you can clearly see down to your scalp when your hair is wet, your hair is thinning, lots of small new hairs sprouting at the hairline or crown is a sign of hair recession.
Holy trinity:
Tabs: https://en.wikipedia.org/wiki/Finasteride
Lotion: https://en.wikipedia.org/wiki/Minoxidil
Shampoo: https://en.wikipedia.org/wiki/Ketoconazole
Finasteride/Dutasteride are 5-alpha reductase inhibitors that prevent free T from being converted to DHT, a byproduct of testosterone that serves no purpose after puberty but to choke out your hair follicles and make them thin out and die in a few short years, a condition known as androgenic alopecia.
5ar inhibitors prevent your hair from getting any worse, mild regrowth is expected, rapid onset diffuse thinners see a significant recovery on fin alone. Can be taken orally or topically.
5ARIs are not dosage dependent, taking as little as as 0.2mg daily fin can reduce serum DHT by 68%, while 5mg reduces by 72%.
0.5mg daily dut reduces serum DHT by 95%, but is considered a second line treatment, and can be more expensive and harder to get prescribed. Start with fin.
Fin has been used to treat benign prostatic hyperplasia (BPH) for 30+ years, it's very well studied and is tolerated well in almost all men, however, there's a 1-2% risk of erectile issues in controlled studies.
These can be avoided by reducing dosage, spacing out dosage, or stopping the drug. Fin takes days to fully bio-accumulate, the half life of fin is about 8 hours, adverse effects occuring within minutes of starting fin or persisting long after stopping fin are not real and have no basis in reality. Please note that dut has a much higher half-life of 5 weeks, if you're prone to fin sides, dut may cause them for far longer.
Refer to https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3481923/ before responding to PFS trolls.
(continued on next post)
Sticky: http://tuxbell.com/index.php/Balding
Reminder that if you can clearly see down to your scalp when your hair is wet, your hair is thinning, lots of small new hairs sprouting at the hairline or crown is a sign of hair recession.
Holy trinity:
Tabs: https://en.wikipedia.org/wiki/Finasteride
Lotion: https://en.wikipedia.org/wiki/Minoxidil
Shampoo: https://en.wikipedia.org/wiki/Ketoconazole
Finasteride/Dutasteride are 5-alpha reductase inhibitors that prevent free T from being converted to DHT, a byproduct of testosterone that serves no purpose after puberty but to choke out your hair follicles and make them thin out and die in a few short years, a condition known as androgenic alopecia.
5ar inhibitors prevent your hair from getting any worse, mild regrowth is expected, rapid onset diffuse thinners see a significant recovery on fin alone. Can be taken orally or topically.
5ARIs are not dosage dependent, taking as little as as 0.2mg daily fin can reduce serum DHT by 68%, while 5mg reduces by 72%.
0.5mg daily dut reduces serum DHT by 95%, but is considered a second line treatment, and can be more expensive and harder to get prescribed. Start with fin.
Fin has been used to treat benign prostatic hyperplasia (BPH) for 30+ years, it's very well studied and is tolerated well in almost all men, however, there's a 1-2% risk of erectile issues in controlled studies.
These can be avoided by reducing dosage, spacing out dosage, or stopping the drug. Fin takes days to fully bio-accumulate, the half life of fin is about 8 hours, adverse effects occuring within minutes of starting fin or persisting long after stopping fin are not real and have no basis in reality. Please note that dut has a much higher half-life of 5 weeks, if you're prone to fin sides, dut may cause them for far longer.
Refer to https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3481923/ before responding to PFS trolls.
(continued on next post)
How common is male pattern baldness in your country?
Hairline doesn't matter.